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mouse microglial cell line bv2 bv2  (Procell Inc)

 
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    Procell Inc mouse microglial cell line bv2 bv2
    Mouse Microglial Cell Line Bv2 Bv2, supplied by Procell Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+microglial+cell+line+bv2+bv2/bv2+cells/pm41819485-68-9-18
    Average 86 stars, based on 1 article reviews
    mouse microglial cell line bv2 bv2 - by Bioz Stars, 2026-09
    86/100 stars

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    Article Title: Ajugol ameliorates mitochondrial dysfunction and cognitive decline in Alzheimer's Disease via BNIP3-dependent mitophagy.
    Article Snippet: Defective mitophagy plays key roles in mitochondrial dysfunction, inflammation, and energy deprivation, and this defect can lead to synaptic loss and cognitive decline in Alzheimer's disease (AD).. Although pharmacological enhancement of mitophagy has been found to ameliorate cognitive impairment in AD models, therapeutic strategies directly targeting this pathway remain limited.. Ajugol, a bioactive iridoid glycoside isolated from Rehmannia glutinosa, has recently been identified as a potential metabolic regulator.



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    In vitro experiments in dopaminergic MN9D cells representing the survival effect of α-syn PFFs after 6h of pyroxamide pre-treatment (A). Survival effect of α-syn PFFs after 3 and 6 hours of pyroxamide pre-treatment in olfactory epithelial T9243 cells (B). Survival effect of α-syn PFFs after 3 hours of pyroxamide pre-treatment in microglial BV2 cells upon α-syn PFFs-induced damage (C). Data were analyzed using one-way ANOVA. * indicates p < 0.05; ** p < 0.01; *** p < 0.001.

    Journal: bioRxiv

    Article Title: Olfactory proteomics reveals the capacity of the HDAC1 inhibitor pyroxamide to halt the α-synuclein preformed fibrils-induced damage in nasal epithelial, microglial and dopaminergic neuronal cell lines

    doi: 10.1101/2025.10.02.679944

    Figure Lengend Snippet: In vitro experiments in dopaminergic MN9D cells representing the survival effect of α-syn PFFs after 6h of pyroxamide pre-treatment (A). Survival effect of α-syn PFFs after 3 and 6 hours of pyroxamide pre-treatment in olfactory epithelial T9243 cells (B). Survival effect of α-syn PFFs after 3 hours of pyroxamide pre-treatment in microglial BV2 cells upon α-syn PFFs-induced damage (C). Data were analyzed using one-way ANOVA. * indicates p < 0.05; ** p < 0.01; *** p < 0.001.

    Article Snippet: Similar conditions were used for the Dopaminergic Neuronal Cell Line (MN9D; Merck Millipore, SCC281), and the Mouse Microglial Cell Line (BV2; Cytion, 305156), seeded at 1 × 10^4 cells/well in 96-well plates.

    Techniques: In Vitro

    Survival analysis associated to pyroxamide pre-treatment 3 hours in dopaminergic neuronal (A) and microglial cells (B) upon oxidative stress conditions. Data were analyzed using one-way ANOVA. * indicates p < 0.05; ** p < 0.01.

    Journal: bioRxiv

    Article Title: Olfactory proteomics reveals the capacity of the HDAC1 inhibitor pyroxamide to halt the α-synuclein preformed fibrils-induced damage in nasal epithelial, microglial and dopaminergic neuronal cell lines

    doi: 10.1101/2025.10.02.679944

    Figure Lengend Snippet: Survival analysis associated to pyroxamide pre-treatment 3 hours in dopaminergic neuronal (A) and microglial cells (B) upon oxidative stress conditions. Data were analyzed using one-way ANOVA. * indicates p < 0.05; ** p < 0.01.

    Article Snippet: Similar conditions were used for the Dopaminergic Neuronal Cell Line (MN9D; Merck Millipore, SCC281), and the Mouse Microglial Cell Line (BV2; Cytion, 305156), seeded at 1 × 10^4 cells/well in 96-well plates.

    Techniques: